Directory
Marta Llovera Tomas

Marta Llovera Tomas

Degree: PhD

973 702 949
marta.llovera(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/B-5585-2009

Publications

  • Zhang, J.; Bahi, N.; Llovera, M.; Comella, J. X.; Sanchis, D.

    Polypyrimidine tract binding proteins (PTB) regulate the expression of apoptotic genes and susceptibility to caspase-dependent apoptosis in differentiating cardiomyocytes.

    CELL DEATH AND DIFFERENTIATION 16 1460-1468. .

    [doi:10.1038/cdd.2009.87]

  • Marler, Katharine J. M.; Becker-Barroso, Elena; Martinez, Albert; Llovera, Marta; Wentzel, Corinna; Poopalasundaram, Subathra; Hindges, Robert; Soriano, Eduardo; Comella, Joan; Drescher, Uwe

    A TrkB/Ephrina Interaction Controls Retinal axon Branching and Synaptogenesis

    JOURNAL OF NEUROSCIENCE 28 12700-12712. .

    [doi:10.1523/JNEUROSCI.1915-08.2008]

  • David, Monica D.; Yeramian, Andree; Dunach, Mireja; Llovera, Marta; Canti, Carles; Garcia de Herreros, Antonio; Comella, Joan X.; Herreros, Judit

    Signalling by neurotrophins and hepatocyte growth factor regulates axon morphogenesis by differential beta-catenin phosphorylation.

    JOURNAL OF CELL SCIENCE 121 2718-2730. .

    [doi:10.1242/jcs.029660]

  • Perez-Garcia, M. Jose; Gou-Fabregas, Myriam; de Pablo, Yolanda; Llovera, Marta; Comella, Joan X.; Soler, Rosa M.

    Neuroprotection by neurotrophic factors and membrane depolarization is regulated by calmodulin kinase IV.

    Journal Of Biological Chemistry 283 4133-4144. .

    [doi:10.1074/jbc.M705477200]

  • Sanchis, Daniel; Llovera, Marta; Ballester, Mane; Comella, Joan X.

    An alternative view of apoptosis in heart development and disease.

    CARDIOVASCULAR RESEARCH 77 448-451. .

    [doi:10.1093/cvr/cvm074]

  • de Pablo, Yolanda; Perez-Garcia, M. Jose; Georgieva, Maya V.; Sanchis, Daniel; Lindqvist, Niclas; Soler, Rosa M.; Comellal, Joan X.; Lloveral, Marta

    Tyr-701 is a new regulatory site for neurotrophin receptor TrkA trafficking and function.

    JOURNAL OF NEUROCHEMISTRY 104 124-139. .

    [doi:10.1111/j.1471-4159.2007.05027.x]

  • Zhang, Jisheng; Bahi, Nuria; Zubiaga, Ana M.; Comella, Joan X.; Llovera, Marta; Sanchis, Daniel

    Developmental silencing and independency from E2F of apoptotic gene expression in postmitotic tissues.

    FEBS LETTERS 581 5781-5786. .

    [doi:10.1016/j.febslet.2007.11.046]

  • Bahi, Nuria; Zhang, Jisheng; Llovera, Marta; Ballester, Manel; Comella, Joan X.; Sanchis, Daniel

    Switch from caspase-dependent to caspase-independent death during heart development: essential role of endonuclease G in ischemia-induced DNA processing of differentiated cardiomyocytes.

    Journal Of Biological Chemistry 281 22943-22952. .

    [doi:10.1074/jbc.M601025200]

  • Benito-Gutierrez, E; Nake, C; Llovera, M; Comella, JX; Garcia-Fernandez, J

    The single amphiTrk receptor highlights increased complexity of neurotrophin signalling in vertebrates and suggests an early role in developing sensory neuroepidermal cells

    Development 132 2191-2202. .

    [doi:10.1242/dev.01803]

  • Perez-Garcia, MJ; Cena, V; de Pablo, Y; Llovera, M; Comella, JX; Soler, RM

    Glial cell line-derived neurotrophic factor increases intracellular calcium concentration. Role of calcium/calmodulin in the activation of the phosphatidylinositol 3-kinase pathway.

    Journal Of Biological Chemistry 279 6132-6142. .

    [doi:10.1074/jbc.M308367200]

  • Llovera, M; de Pablo, Y; Egea, J; Encinas, M; Peiro, S; Martin-Zanca, D; Rocamora, N; Comella, JX

    Trk is a calmodulin-binding protein: implications for receptor processing.

    JOURNAL OF NEUROCHEMISTRY 88 422-433. .

    [doi:10.1046/j.1471-4159.2003.02178.x]

  • Dominguez, C; Moreno, A; Llovera, M

    Aluminum ions induce DNa synthesis but not cell proliferation in human fibroblasts in vitro

    BIOL TRACE ELEM RES 86 1-10. .

    [doi:10.1385/BTER:86:1:01]

Projects

  • INVESTIGO 2022: Senyalització cel·lular i apoptosi
  • New molecular functions of apoptotic genes in cardiac development and stress
  • ANALISIS DEL POTENCIAL DE LOS INHBIDORES DE LAS HISTONA DESACETILASAS EN LA SENSIBILIZACION A LA MUERTE POR TRAIL DE BIOBLASTOMAS HUMANOS