 
            Anna Maria Casanovas Llorens
Degree: PhD
                                
                ana.casanovas(ELIMINAR)@udl.cat
            
ResearcherID: http://www.researcherid.com/rid/C-3984-2011
Publications
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                        Caldero, Jordi; Tarabal, Olga; Casanovas, Anna; Ciutat, Dolors; Casas, Celia; Llado, Jeronia; Esquerda, Josep E. Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation.Journal Of Neuroscience Research 85 2726-2740. . 
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                        Serrando, M; CASANOVAS, A; ESQUERDA, JE Occurrence of glutamate receptor subunit 1-containing aggresome-like structures during normal development of rat spinal cord interneurons.Journal Of Comparative Neurology 442 23-34. . 
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                        CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; ESQUERDA, JE c-Jun regulation in rat neonatal motoneurons postaxotomy.Journal Of Neuroscience Research 63 469-479. . 
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                        CASANOVAS, A; Olmos, G; Ribera, J; Boronat, MA; ESQUERDA, JE; Garcia-Sevilla, JA Induction of reactive astrocytosis and prevention of motoneuron cell death by the I(2)-imidazoline receptor ligand LSL 60101.BRITISH JOURNAL OF PHARMACOLOGY 130 1767-1776. . 
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                        Tarabal, O; LladoJ, OJ; CALDERO, J; Ribera, J; CIUTAT, D; CASANOVAS, A; Ayala, V; Serrando, M; Casas, C; Sarasa, M; ESQUERDA, JE Chronic NMDa treatment results in a prevention of acute excitotoxic injury at tm chick embryo motoneurons and induces permanent changes in intracellular membrane compartimentation.EUROPEAN JOURNAL OF NEUROSCIENCE 12 38-38. . 
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                        CALDERO, J; Llado, J; Ribera, J; Tarabal, O; CIUTAT, D; CASANOVAS, A; Ayala, V; Casas, C; Serrando, M; Oppenheim, RW; ESQUERDA, JE Chronic administration of n-methyl-d-aspartate (NMDa) prevents excitotoxicity and normal and induced programmed cell death in spinal cord motoneurons.EUROPEAN JOURNAL OF NEUROSCIENCE 12 394-394. . 
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                        CASANOVAS, A; Ribera, J; Hager, G; Kreutzberg, GW; Casas, C; Ayala, V; CALDERO, J; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE Differential expression of c-jun after axotomy in neonatals suggest different functions in programmed cell death and axonal regeneration.EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. . 
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                        Casas, C; Ayala, V; Ribera, J; CALDERO, J; CASANOVAS, A; CIUTAT, D; Tarabal, O; Llado, J; Serrando, M; ESQUERDA, JE c-jun-like immunoreactivity specifically associated with apoptosis reveals new epitopes emerged by caspase-3 cleavage.EUROPEAN JOURNAL OF NEUROSCIENCE 12 8-8. . 
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                        Ribera, J; Marsal, J; CASANOVAS, A; Hukkanen, M; Tarabal, O; ESQUERDA, JE Nitric oxide synthase in rat neuromuscular junctions and in nerve terminals of Torpedo electric organ: its role as regulator of acetylcholine release.Journal Of Neuroscience Research 51 90-102. . [doi:10.1002/(SICI)1097-4547(19980101)51:1<90::AID-JNR10>3.0.CO;2-C] 
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                        CASANOVAS, A; Ribera, J; Hukkanen, M; RiverosMoreno, V; ESQUERDA, JE Prevention by lamotrigine, MK-801 and N-omega-nitro-L-arginine methyl ester of motoneuron cell death after neonatal axotomyNEUROSCIENCE 71 313-325. . 
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                        Casanovas A; Ribera J; Hukkanen M; Riveros-Moreno V; Esquerda JE Prevention by lamotrigine, MK-801 and N omega-nitro-L-arginine methyl ester of motoneuron cell death after neonatal axotomy.NEUROSCIENCE 71 313-325. . 
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                        CALDERO, J; CASANOVAS, A; SORRIBAS, A; ESQUERDA, JE CaLCITONIN GENE-RELaTED PEPTIDE IN RaT SPINaL-CORD MOTONEURONS - SUBCELLULaR-DISTRIBUTION aND CHaNGES INDUCED BY aXOTOMYNEUROSCIENCE 48 449-461. . 
Projects
- Preclinical Analisis of new combinatorial treatments for spinal muscular atrophy (SMA): Effects on motorneuron survival, synaptic integrity, and skeletal muscle preservation.
- Los aferentes sinápticos de tipo C en las motoneuronas: implicaciones en la fisiopatología y terapia de la esclerosis lateral amiotrófica y de la atrofia muscular espinal (Motoneurona)
- Mecanismes excitotòxics, resposta neuroinflamatòria i factors sèrics en l´esclerosi lateral amiotròfica humana i experimental