Directorio de personas
Elisa Cabiscol Catala

Elisa Cabiscol Catala

Grado: Doctor/a

973 702 281
elisa.cabiscol(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/A-4584-2009

Publicaciones

  • Gomez-Pastor, Rocio; Perez-Torrado, Roberto; Cabiscol, Elisa; Matallana, Emilia

    Transcriptomic and proteomic insights of the wine yeast biomass propagation process

    FEMS YEAST RESEARCH 10 870-884. .

    [doi:10.1111/j.1567-1364.2010.00667.x]

  • Alba Sorolla, Ma; Jose Rodriguez-Colman, Ma; Tamarit, Jordi; Ortega, Zaira; Lucas, Jose J.; Ferrer, Isidre; Ros, Joaquim; Cabiscol, Elisa

    Protein oxidation in Huntington disease affects energy production and vitamin B6 metabolism.

    FREE RADICAL BIOLOGY AND MEDICINE 49 612-621. .

    [doi:10.1016/j.freeradbiomed.2010.05.016]

  • Gomez-Pastor, Rocio; Perez-Torrado, Roberto; Cabiscol, Elisa; Ros, Joaquim; Matallana, Emilia

    Reduction of oxidative cellular damage by overexpression of the thioredoxin TRX2 gene improves yield and quality of wine yeast dry active biomass.

    MICROBIAL CELL FACTORIES 9 9-9. .

    [doi:10.1186/1475-2859-9-9]

  • Irazusta, Veronica; Obis, Elia; Moreno-Cermeno, Armando; Cabiscol, Elisa; Ros, Joaquim; Tamarit, Jordi

    Yeast frataxin mutants display decreased superoxide dismutase activity crucial to promote protein oxidative damage.

    FREE RADICAL BIOLOGY AND MEDICINE 48 411-420. .

    [doi:10.1016/j.freeradbiomed.2009.11.010]

  • Herrero, Enrique; Ros, Joaquim; Belli, Gemma; Cabiscol, Elisa

    Redox control and oxidative stress in yeast cells.

    BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS 1780 1217-1235. .

    [doi:10.1016/j.bbagen.2007.12.004]

  • Sorolla, M. Alba; Reverter-Branchat, Gemma; Tamarit, Jordi; Ferrer, Isidre; Ros, Joaquim; Cabiscol, Elisa

    Proteomic and oxidative stress analysis in human brain samples of Huntington disease.

    FREE RADICAL BIOLOGY AND MEDICINE 45 667-678. .

    [doi:10.1016/j.freeradbiomed.2008.05.014]

  • Irazusta, Veronica; Moreno-Cermeno, Armando; Cabiscol, Elisa; Ros, Joaquim; Tamarit, Jordi

    Major targets of iron-induced protein oxidative damage in frataxin-deficient yeasts are magnesium-binding proteins.

    FREE RADICAL BIOLOGY AND MEDICINE 44 1712-1723. .

    [doi:10.1016/j.freeradbiomed.2008.01.014]

  • Reverter-Branchat, Gemma; Cabiscol, Elisa; Tamarit, Jordi; Sorolla, M. Alba; de la Torre, M. Angeles; Ros, Joaquim

    Chronological and replicative life-span extension in Saccharomyces cerevisiae by increased dosage of alcohol dehydrogenase 1.

    MICROBIOLOGY-SGM 153 3667-3676. .

    [doi:10.1099/mic.0.2007/009340-0]

  • Irazusta, V; Cabiscol, E; Reverter-Branchat, G; Ros, J; Tamarit, J

    Manganese is the link between frataxin and iron-sulfur deficiency in the yeast model of Friedreich ataxia.

    Journal Of Biological Chemistry 281 12227-12232. .

    [doi:10.1074/jbc.M511649200]

  • Reverter-Branchat, G; Cabiscol, E; Tamarit, J; Ros, J

    Oxidative damage to specific proteins in replicative and chronological-aged Saccharomyces cerevisiae: common targets and prevention by calorie restriction.

    Journal Of Biological Chemistry 279 31983-31989. .

    [doi:10.1074/jbc.M404849200]

  • Echave, P; Tamarit, J; Cabiscol, E; Ros, J

    Novel antioxidant role of alcohol dehydrogenase E from Escherichia coli.

    Journal Of Biological Chemistry 278 30193-30198. .

    [doi:10.1074/jbc.M304351200]

  • Tamarit, J; Belli, G; Cabiscol, E; Herrero, E; Ros, J

    Biochemical characterization of yeast mitochondrial Grx5 monothiol glutaredoxin.

    Journal Of Biological Chemistry 278 25745-25751. .

    [doi:10.1074/jbc.M303477200]

Proyectos

  • Uncovering the function of a conserved site in eukaryotic frataxins.
  • PATHOPHYSIOLOGY AND THERAPEUTICS OF FRIENDREICH ATAXIA: MARKERS OR OXIDATIVE STRESS FROM YEAST TO CARDIOMYOCYTES
  • Papel del factor de transcripción Hcm1 en la biogénesis mitocondrial, envejecimiento y la limitación de nutrientes en S. cervisiae
  • Papel funcional del estrés oxidativo y nitrosativo en grandes sistemas biológicos.
  • Estrés oxidativo, envejecimiento celular, homeostasis del hierro y patologías relacionadas en S. cerevisae como modelo celular