Directorio de personas
Jordi Tamarit Sumalla

Jordi Tamarit Sumalla

Grado: Doctor/a

973 702 475
jordi.tamarit(ELIMINAR)@udl.cat

ResearcherID: http://www.researcherid.com/rid/C-6316-2008

Publicaciones

  • Alsina, D; Ros, J; Tamarit, J

    Nitric oxide prevents Aft1 activation and metabolic remodeling in frataxin-deficient yeast

    Redox Biology 14 131-141. .

    [doi:10.1016/j.redox.2017.09.001]

  • Britti, E; Delaspre, F; Feldman, A; Osborne, M; Greif, H; Tamarit, J; Ros, J

    Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment

    Journal Of Cellular And Molecular Medicine 22 834-848. .

    [doi:10.1111/jcmm.13365]

  • Purroy, R; Britti, E; Delaspre, F; Tamarit, J; Ros, J

    Mitochondrial pore opening and loss of Ca2+ exchanger NCLX levels occur after frataxin depletion

    BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE 1864 618-631. .

    [doi:10.1016/j.bbadis.2017.12.005]

  • Cabiscol E.; Tamarit J.; Ros J.

    Specificity of Protein Carbonylation and Its Relevance in Aging

    Protein Carbonylation: Principles, Analysis, And Biological Implications 340-383. .

    [doi:10.1002/9781119374947.ch14]

  • Tamarit, J; Obis È; Ros, J

    Oxidative stress and altered lipid metabolism in Friedreich ataxia

    FREE RADICAL BIOLOGY AND MEDICINE 100 138-146. .

    [doi:10.1016/j.freeradbiomed.2016.06.007]

  • Obis E; Irazusta V; Sanchís D; Ros J; Tamarit J

    Frataxin deficiency in neonatal rat ventricular myocytes targets mitochondria and lipid metabolism

    FREE RADICAL BIOLOGY AND MEDICINE 73 21-33. .

    [doi:10.1016/j.freeradbiomed.2014.04.016]

  • Mincheva-Tasheva S; Obis E; Tamarit J; Ros J

    Apoptotic cell death and altered calcium homeostasis caused by frataxin depletion in dorsal root ganglia neurons can be prevented by BH4 domain of Bcl-x(L) protein

    HUMAN MOLECULAR GENETICS 23 1829-1841. .

    [doi:10.1093/hmg/ddt576]

  • Cabiscol E; Tamarit J; Ros J

    PROTEIN CARBONYLATION: PROTEOMICS, SPECIFICITY AND RELEVANCE TO AGING

    MASS SPECTROMETRY REVIEWS 33 21-48. .

    [doi:10.1002/mas.21375]

  • Moreno-Cermeño A; Alsina D; Cabiscol E; Tamarit J; Ros J

    Metabolic remodeling in frataxin-deficient yeast is mediated by Cth2 and Adr1

    BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH 1833 3326-3337. .

    [doi:10.1016/j.bbamcr.2013.09.019]

  • Rodríguez-Colman MJ; Sorolla MA; Vall-Llaura N; Tamarit J; Ros J; Cabiscol E

    The FOX transcription factor Hcm1 regulates oxidative metabolism in response to early nutrient limitation in yeast. Role of Snf1 and Tor1/Sch9 kinases

    BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH 1833 2004-2015. .

    [doi:10.1016/j.bbamcr.2013.02.015]

  • Tamarit, Jordi; de Hoogh, Anouk; Obis, Elia; Alsina, David; Cabiscol, Elisa; Ros, Joaquim

    Analysis of oxidative stress-induced protein carbonylation using fluorescent hydrazides

    JOURNAL OF PROTEOMICS 75 3778-3788. .

    [doi:10.1016/j.jprot.2012.04.046]

  • Sorolla, M. Alba; Jose Rodriguez-Colman, Maria; Vall-llaura, Nuria; Tamarit, Jordi; Ros, Joaquim; Cabiscol, Elisa

    Protein oxidation in Huntington disease

    BIOFACTORS 38 173-185. .

    [doi:10.1002/biof.1013]

Proyectos

  • Uncovering the function of a conserved site in eukaryotic frataxins.
  • Mecanismos de disfuncion mitocondrial y nuevas estrategias terapéuticas en modelos celulares y animales de Ataxia de Friedreich
  • Nuevo fármaco dirigido a la Ataxia de Friedreich
  • Targeting mitochondria in Friedreich Ataxia: molecular mechanisms and therapeutic approaches
  • MODELOS CELULARES DE ATAXIA DE FRIEDREICH: BASES MOLECULARES DE LA PATOLOGIA E IDENTIFICACION DE BIOMARCADORES Y COMPUESTOS CON POTENCIAL TERAPEUTICO
  • Modelos celulares de ataxia de Friedreich: bases moleculares de la patología e identificación de biomarcadores y compuestos con potencial terapéutico
  • PATHOPHYSIOLOGY AND THERAPEUTICS OF FRIENDREICH ATAXIA: MARKERS OR OXIDATIVE STRESS FROM YEAST TO CARDIOMYOCYTES
  • Papel funcional del estrés oxidativo y nitrosativo en grandes sistemas biológicos.
  • Estrés oxidativo, envejecimiento celular, homeostasis del hierro y patologías relacionadas en S. cerevisae como modelo celular